Unit 4: Cell Communication & Cycle
Biology · Unit 4 · Paper 2

Cell Communication & Cycle unit test

A test on this unit alone, marked as a percentage and a letter grade — for the test your class is actually sitting, rather than for May. Answer everything, then submit once: seeing the answer to question 3 before attempting question 4 makes the final percentage meaningless.

Each paper is built from this unit’s 37 terms and is the same for everyone, so a teacher can assign “Unit 4, Paper 2” and every student sits the identical test. Multiple choice is marked objectively; the written sections you mark yourself against the model answer and rubric.
Suggested time 36 min 33 points0/17 attempted
1

Second messenger

2

Cancer as checkpoint failure

3

Meiosis I vs meiosis II

4

Receptor specificity

5

Calcium as a second messenger

6

Centrosome and spindle

7

Sister chromatids vs homologous chromosomes

8

Meiosis vs mitosis

9

cAMP pathway

10

Apoptosis

11

Signal termination

12

Cytokinesis

Short answer 1. Define or explain: Synapsis and tetrads

3 pts

Short answer 2. Define or explain: G1 checkpoint

3 pts

Short answer 3. Define or explain: Negative and positive feedback

3 pts

Short answer 4. Define or explain: Lab: Mitosis and Meiosis

3 pts

Free response

9 pts

A growth-factor signaling pathway in epithelial cells works as follows: an extracellular growth factor binds a receptor tyrosine kinase, which activates a series of relay kinases in the cytoplasm; the last kinase phosphorylates a transcription factor that turns on genes promoting progression through the cell cycle. A tumor is found to carry a receptor mutation that locks the receptor in its active conformation even when no growth factor is present.

Describe the three general stages of signal transduction as they apply to this pathway.

Explain how a phosphorylation cascade amplifies the original signal.

Predict the effect of the receptor mutation on cell division in this tissue and justify your prediction.

A drug inhibits a relay kinase downstream of the receptor. Predict whether it would slow the growth of this tumor and explain.

Explain why a drug designed to occupy the growth-factor binding site of the receptor would be ineffective against this particular tumor.